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Oxidative stress plays an important role in the pathogenesis of all types of cataracts

[3][4] Research has examined T4 (and its TB-500 fragment) in the context of: Cell migration at wound margins Corneal and dermal wound healing in preclinical models [5] Cardiac repair following ischemic injury an area of active investigation Hair follicle stem cell migration Anti-inflammatory modulation The active 17-amino-acid sequence is well-characterized, and synthetic TB-500 is widely used in research as a stable proxy for full-length T4 protein

Studies in PCa suggest that arginine derived metabolites promote tumor proliferation by suppressing apoptosis through redox modulation (49)

Oxidative stress promotes myofibroblast differentiation and tumour spreading