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In addition, HCs and KTRs controlling CMV infection responded with a stronger upregulation of G6PD and CD98 than noncontrollers (Figure 4C)

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Furthermore, the study hypothesizes a potential interaction of AOD9604 with the beta-adrenergic pathway, especially concerning the beta(3)-adrenergic receptors (beta(3)-AR), which are considered to be key lipolytic receptors found in fat cells
