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Description
By slowing gastric emptying, it reduces the time stomach acid has to work on B12-protein bonds
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If the abovementioned is occurring, it could mean that IGF-1 administration could also lower glucose levels by suppressing hepatic gluconeogenesis, as well as improving insulin signalling in this organ, leading to IGFBP-1 suppression and overall improvement of the IGF-1/GH/insulin axis
These changes are slower by nature, so patience helps here
