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While autophagy suppresses apoptosis and pyroptosis by degrading Caspase-8 [162], excessive autophagy (autophagic cell death) releases unprocessed autophagosomes containing free fatty acids and ROS (O 2 - , H 2 O 2 ), activating RIPK1-dependent necroptosis [163]
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In general, elevated ROS reduces RBC survivability in vivo 44 , suggesting this could be an additional contributor to pathogenesis of bystander effect in malaria
