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Increased oxidative stress due to smoking contributes to atherosclerosis by inducing cell proliferation, cell hypertrophy, and apoptosis, either directly through vascular endothelial damage or through activation of various intracellular signaling pathways

168 In lung cancer, the inhibition of DNMT1 can reduce cell proliferation and increase apoptosis, and its activity is related to the invasiveness and metastatic potential of tumors

[] So, VitE may have a protective role in the long-term complications of chronic hemolysis such as pigment gallstone and renal dysfunction with chronic hemoglobin sedimentation in the kidney

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