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The mechanism behind symptom onset is represented by overstimulation of D2 dopaminergic receptors and neuronal oxidative stress, which destabilizes synaptic homeostasis, favoring excitotoxicity and microglial inflammation
This is called angiogenesis
Chronic hemolytic anemia due to glucose-6-phosphate dehydrogenase deficiency or glutathione synthetase deficiency: the role of vitamin E in its treatment
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