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However, several studies revealed that independent of c-MET signaling, tivantinib also disrupted microtubule dynamics, induced G2/M arrest and apoptosis, overcame ABC transportermediated multidrug-resistant, and acted as a microtubule polymerization inhibitor by binding to tubulin via the colchicine-binding site (Basilico et al., 2013
Mech Ageing Dev 41:2938
Researchers have shown that it raises glutathione levels
