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In humans, only GLRX3 (also called PICOT) is found in the cytosol where it interacts with the cytosolic form of the BOLA family in humans, BOLA2 [129, 169]

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Increasing evidence suggests that Cu dysregulation, including Cu excess, Cu deficiency, and uneven Cu distribution, promotes cell dysfunction and death by disrupting antioxidant systems, cuproptosis, insulin secretion, and glucose metabolism, thereby significantly regulating the development of DM and its complications (5558)

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