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Abstract Clinical and experimental data hints that prolonged and repeated epileptic seizures can lead to molecular, biochemical, metabolic, and structural changes in the brain, a continuous process of chronic brain injury that ultimately leads to neuronal death

In comparison to prior narrative syntheses that propose putative mechanisms without systematically evaluating lipid pathways, the present review advances the field by integrating findings from experimental, preclinical, and translational studies to map how UPF consumption alters lipid synthesis, transport, and signaling within the central nervous system

doi: 10.1093/bib/bbae350

This autoimmune response negatively affects neurological functioning and triggers an often rapid, acute onset of Obsessive Compulsive Disorder (OCD), anxiety, restricted food intake, tics and other symptoms, which can develop either very quickly following the illness or have a delay of many months after infection