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Metabolism abnormalities induced by exposure to excessive UA are important factors mediating the pathological development of HUA-associated multiple organ complications ( via UA excretion agent benzbromarone therapy ( via regulating transforming growth factor 1, nicotinamide adenine dinucleotide phosphate oxidase and AMP-activated protein kinase mediated reactive oxygen species and inflammation (Madlala et al., 2016
About this article Cite this article Odendaal, C., Jager, E.A., Martines, AC.M.F
Neuroprotective effect of STAZN, a novel azulenyl nitrone antioxidant, in focal cerebral ischemia in rats: dose-Response and therapeutic window
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