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Accordingly, an improved understanding in humans of the microbial pathways involved in TMA/TMAO formation from distinct nutrients is an essential and necessary step in the development of targeted interventions to interrupt these processes

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With the induction of the NF-B signaling pathway in HUVECs, the subsequent upregulation of expression of vascular cell adhesion molecule-1 (VCAM-1) and intercellular adhesion molecule-1 (ICAM-1) enhanced the adhesion of monocytes to the vascular cells (84)