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Diehl, L
[23] The ALDH2-/- mice display age-related memory deficits in various tasks, as well as endothelial dysfunction, brain atrophy, and other Alzheimer's disease-associated pathologies, including marked increases in lipid peroxidation products, amyloid-beta, p-tau and activated caspases
This gives the peptide enough time to initiate healing cascades and allow the body to continue repairing even after the peptide is no longer present
Disease-associated oligodendrocyte responses across neurodegenerative diseases
