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This review systematically elucidates how the microbiota-ENS axis modulates the function of the ENS through multilevel signaling transduction involving microbial metabolites (SCFAs, BAs), intestinal hormones (GLP-1, CCK, leptin, ghrelin), and neurotransmitters (5-HT, Ach, VIP, NO, GABA), thereby regulating gastrointestinal motility

In the present work, we investigated the circulatory half-life of the bispecific HSA constructs in an hFcRn +/+ , hAlb +/+ AlbuMus model 27

Stimulation with lipopolysaccharide (LPS) and IFN not only activated M0 macrophages into M1 phenotype but also upregulated the expression of inducible nitric oxide synthase (iNOS) in Gpx4-deficient macrophages

PMID 22387672