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Naunyn-Schmiedebergs Archives of Pharmacology
By transferring mitochondria from Parkinsons patients into mtDNA-depleted cells that were otherwise healthy, researchers identified defects in the electron transport chain and heightened oxidative stress, underscoring the role of mtDNA alterations in Parkinsons disease (Miller et al., 1996
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This highly flexible arrangement facilitates simultaneous binding of several host endocytic proteins and modulation of the hosts PM (Fig
