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*Correspondence: Xingsheng Wang, [email protected] Lai, [email protected] These authors have contributed equally to this work Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Long-term outcomes in FLT3-mutated acute myeloid leukemia after frontline hypomethylating agent, venetoclax and a FLT3 inhibitor

Based on the findings from the enrichment functional analysis of miRNA, it can be inferred that the pathway in question may be subject to regulation by hsa-mir-2964-5p, has-mir-412-5p, hsa-mir-4536, mir-704, and has-mir-187 in the context of sperm

When this happens alongside a steep decline in NAD+, cells lose both the engine that powers repair and the shield that blocks damage