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TSCI pathogenesis is characterized by two distinct phases: the primary injury, which results in irreversible mechanical damage, and the secondary injury, driven by cellular membrane disruption, apoptotic signaling, and ischemia caused by microvascular compromise [4, 5]
A structured maintenance plan is recommended
As an extending point, there appears to be fistula healing (i.e., simultaneous healing of the skin and other tissues wounds) (Klicek et al., 2008
The Buthionine sulfoximine (BSO) irreversibly binds to -GCS, which is required during the condensation of glutamate and cysteine to produce GSH
