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Specifically, mitochondria have been linked to oxidative stress and subsequent inflammatory processes in a wide range of pathological conditions (Table 3, suggested a deficiency in oxidative stress defense as well as an increased lipid peroxidation, results already reported in FM and even linked to pain intensity and inflammation in those patients (Meeus et al., 2013
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CRC Press, Boca Raton, FL, pp 83108 Spencer JP, Rice-Evans C, Williams RJ (2003) Modulation of pro-survival Akt/protein kinase B and ERK1/2 signaling cascades by quercetin and its in vivo metabolites underlie their action on neuronal viability
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