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Hence, it can be seen that many environmental pollutants are capable of inducing eryptosis in human RBCs, and that eryptosis may be an effective indicator for comparing the hematotoxicity of parent compounds and their metabolites [423], to assess the toxicity of their potential replacements [421, 422, 428], and to investigate the mechanisms of action of toxic metals [184, 420, 424, 435] and pesticides [436, 437]

In this experiment Schwarzlnder and co-workers (2009) demonstrated a redox perturbation in the mitochondria after Cd exposure with important effects on redox signaling

[1] [3] If left untreated, some of these changes can become permanent

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