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allopurinol, with its exquisite mechanistic precision, conducts a targeted biochemical silencing of xanthine oxidase enzymatic networks, fundamentally disrupting the pathophysiological cascade responsible for uric acid generation (Shuvo et al., 2025)
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Given that organelles orchestrate essential intracellular processes, further elucidation of how 4-HNE impairs organelle function in cardiomyocytes may yield novel mechanistic insights and advance therapeutic strategies
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