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On the contrary, L-carnitine administration significantly changed maximal oxygen consumption (VO 2 ) at rest ( p 50% was considered statistically significant between the included studies [18]
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Recent studies have shown that the death-associated protein kinase DRAK2 contributes to the pathogenesis of NAFLD and NASH by disrupting the alternative splicing of mitochondrial-related genes, including mitochondrial DNA polymerase POL2, through the RNA splicing factor SRSF6 [288]
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