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Currently, cellular senescence is understood as the response of cells to stress induced by both endogenous and exogenous factors, such as oncogenic factors, and genotoxic stress, telomere dysfunction, or mitochondrial degradation [11]

2012;63(Suppl 1):829

Inflammation is part of tissue response, but excessive or poorly regulated inflammatory signalling can interfere with repair processes

The plethora of structural information, together with the increasing power of computational facilities and refinement of binding prediction tools (110, 112, 119), open the possibility to design peptides/peptidomimetics able to target the translation complexes, with the aim to decrease protein synthesis of specific neuronal mRNAs and rescuing a healthy phenotype in individuals affected by FXS