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Antioxidant capacity in the lung is substantially reduced and oxidative stress can persist long after the cessation of cigarette smoking or an acute exacerbation of COPD owing to continued production of ROS from endogenous sources [9, 12]
have more recently demonstrated Fas activation-induced eryptosis via ceramide formation in human erythrocytes exposed to CSE, as reported in the relevant paragraph [130]
In: Motulsky, A.G., Bobrow, M., Harper, P.S., Schivver, C
