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Conclusions Astrogliopathology is complex, heterogeneous and context dependent with regard to disorder, disorder-stage, comorbidities, age, and sex
Our findings demonstrate that M-SYFSY effectively reverses pathological metabolic reprogramming by inhibiting HIF-1-mediated transcriptional activation of key glycolytic enzymes including HK2, PDK1, and LDH
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As described for light microscopy, we found little colocalization of GPX4 with TH in putamen samples from non-PD subjects
