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Description
Despite normal -cell replication, apoptosis is reduced, which ultimately decreases the number of cells by 50% [3]
synchronously regulates the wound microenvironment, significantly eliminating DPPH free radicals
This oxidative stress damages mitochondrial components, including lipids, proteins, and mitochondrial DNA (mtDNA) [177]
In accordance with theory of hormesis, we suppose that such mild, repeated stresses are responsible for adaptation of rats to subsequent stresses, e.g
