glutathione copper toxicity homeostasis and copper-induced cell death in the pathogenesis of cardiovascular disease and therapeutic strategies Glutathione metabolism is conserved in
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Second, the doses are small because the target is a high-affinity intracellular enzyme, not a receptor that needs saturating with milligrams of drug
Wang Y, Wang X, Fu H, Kou S, Huang D, Shen Z, et al
In our previous study, we observed a significant increase in GRP78 immunofluorescence labeling in 12Z cells following NAC treatment 20 , suggesting an association between NAC and organelle stress responses
10.1016/j.pain.2005.02.006 92 LiponeP.EhlerE.NastajM.Palka-KisielowskaI.CruccuG.TruiniA.et al (2020)
