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Timing and administration studies: Research has also explored optimal timing for AOD9604 dosing

Although hypothetical, differential CD38 hydrolase inhibition by anti-CD38 antibodies might result in divergent abilities to suppress the formation of ADO

According to our data, this resistance may persist despite CD38 downregulation, likely due to sustained production of micromolar concentrations of immunosuppressive ADO detected in patient samples at progression and following DARA failure

However, directly comparing their efficacy is challenging due to differences in study designs, patient populations, and treatment durations