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Such dysregulated microbiota-host interactions may disrupt immune homeostasis by affecting iron metabolism and subsequently reshape the bodys immune tolerance state by driving the differentiation of Tregs

These agents have the ability to interrupt microbial membranes and hence can be candidates for treating various infectious diseases, including infections resistant to antibiotics [144]

Collectively, these findings demonstrate that diet is a potent modulator of the human methylome and contributes to both short-term metabolic regulation and long-term health trajectories
