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HSV-1 infection can also promote neuroinflammation by triggering the type 1 interferon response
DSIP has been shown in various studies to interact with these neurotransmitter systems, though whether this represents direct receptor binding, allosteric modulation, or indirect effects on neurotransmitter synthesis, release, or metabolism remains unclear
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Enhancement of Xrcc1-mediated base excision repair improves the genetic stability and pluripotency of iPSCs
