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Using this mechanistic model of bile acids, we explain how abnormal bile acid physiology drives disease pathogenesis, emphasizing how ceramide synthesis may serve as a unifying pathogenic feature for cardiometabolic diseases

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Antioxidant Glutathione Inhibits Inflammation in Synovial Fibroblasts via PTEN/PI3K/AKT Pathway: An in Vitro Study. Archives of Rheumatology :/Archives of Rheumatology, vol

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