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Several reports (Table 1) have used other parts of A

Recent studies have shown that HSCs have unique metabolic characteristics
Evidence suggests multiple interrelated pathways of liver injury: Nicotine-induced injury : Nicotine is metabolized in the liver via CYP2A6, where it activates 7-nicotinic acetylcholine receptors (7-nAChR), leading to oxidative stress, mitochondrial dysfunction, and pro-fibrotic signaling [36, 125, 126]

However, only two pH values were evaluated, so no specific conclusion could be drawn from those studies [25]
