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To confirm that the mechanism of protection was dependent, at least in part, on enhanced GSH synthesis, CGNs were cotreated with Immunocal and the -glutamyl-cysteine ligase inhibitor, BSO, which prevents GSH synthesis [39]
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Endothelium-specific CYP2J2 overexpression improves cardiac dysfunction by promoting angiogenesis via Jagged1/Notch1 signaling
Pappa, S
