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Description
BPC-157 appears to work through tissue repair mechanisms rather than inflammation suppression, and the human study data shows better durability at 12 months

K-Hcy modification drives nuclear-to-cytoplasmic translocation and extracellular release of HMGB1/2, thereby amplifying paracrine senescence signaling through enhanced SASP

TT: Writing original draft
doi: 10.1080/10408398.2020.1803200
