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Upregulation of type I IFN in SLE is thought to induce an IFN epigenomic signature, activating latent enhancers and bookmarking chromatin, reprogramming genes to be hyper-responsive, amplifying the inflammatory cascade (146148)

Quantitative metabolite profiling of the TIF and serum human patient samples was performed based on a protocol adapted from a previous study 28

Cytosolic GSTs are classified on the basis of sequence similarities and structural properties

This study could not achieve full blinding due to the nature of the interventionreduced glutathione requires intravenous administration, while the control group only received oral entecavir therapy