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OS provides one explanation for the pathogenesis of preeclampsia since it leads to lipid peroxidation accompanied by endothelial dysfunction, i.e., a consequence of periods of ischemiareperfusion generated from failed spiral artery remodelling during placentation

Numerous investigations on c-MET aberrant signaling provide a strong rationale for exploring the therapeutic potential of specific receptor antagonists

herpochory, or crawling with fine hair-like structures called trichomes

The reduced gene expression or deletion of MET in vivo in mice results in altered synapse maturation (reviewed in Ma and Qiu, 2019)
