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These activation mechanisms also result in the creation of pro-inflammatory mediators and adhesion molecules at the cell surface, which worsen the pro-inflammatory and oxidant environment
At the mitochondrial level, using copper chelators can lead to a decrease in COX levels and mitochondrial respiration levels and activate glycolysis (36, 38)
Hydrogels that mimic developmentally relevant matrix and N-cadherin interactions enhance MSC chondrogenesis
Clinical and biological double-blind-study of ticlopidine in preventive treatment of sickle-cell disease crises
