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Huai et al
Therefore, the ensuing discussion primarily focuses on CIN, specifically examining how these metabolic vulnerabilities drive DNA damage accumulation, exacerbate replication stress, and compromise specific DNA repair pathways
doi: 10.1016/s0076-6879(02)53072-x
Changes in intestinal and hepatic thyroid hormone deiodination during spontaneous metamorphosis of the sea lamprey, Petromyzon marinus
