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Similarly, paclitaxel binds with -tubulin, the main component of the mitochondrial membrane, and opens the mitochondrial permeability transition pore (mPTP), resulting in a rapid mitochondrial depolarization and cytochrome c release from mitochondria in a neuroblastoma cell line ( in vitro studies, paclitaxel-induced mitochondrial dysfunction significantly hampered cellular respiratory function and ATP production in DRG neurons (Ikegami and Koike, 2003

Nat Rev Clin Oncol 2021;18(10):663672

Like they would with any other healthcare offering, consumers should approach peptide clinics with a healthy degree of scrutiny

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