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NNMT is a known drain on NAD+, and 5-Amino-1MQ appears to inhibit this process by helping maintain more usable NAD+ within cells

Heres the whole thing in plain terms: What they did: built and validated a lab-grade method to accurately measure 5A1MQ in the blood, then dosed rats two ways intravenous (IV) and oral and tracked exactly how much showed up in circulation over time

They are also supported by the finding that long-term administration of recombinant IGF-1 in humans with GH receptor deficiency is associated with decreased fat mass, increased lipolysis, and a faster rate of lipid oxidation (116)

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