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Thus, blocking FAO is a potential therapeutic strategy for impairing energy production in cancer cells

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Because phosphorylated signal transducer and activator of transcription (STAT) 3 (pSer727) was also increased in the resistant HCC cells, treatment with a small-molecule STAT inhibitor reverted the phosphorylation and subsequent mitochondrial protein alterations, enhancing sensitivity to sorafenib (Pandit et al., 2021)

Each hormone is a product of posttranslational modification of the POMC gene transcript and contains the sequence of His-Phe-Arg-Trp, considered to be the core of agonist activity [35, 36]