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Abstract Researchers have implicated mercury (Hg) exposure in the pathogenesis of autism spectrum disorder (ASD) through multiple mechanisms, including oxidative stress, mitochondrial dysfunction, and glutamatergic dysregulation
its ambiguous effect, since it is also used therapeutically, depends on two distinct mechanisms: the NF-kB signaling pathway is responsible for the therapeutic effects, whereas the AP-1 pathway is responsible for the pathological effects
