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Program in Sciences and Innovation in Medicine at Universidad del Desarrollo The PIKfyve-ULK1 Axis: Coordinating Lysosomal Membrane Repair and Mitochondrial Bioenergetics Disruption of the PIKfyve/Fig4/Vac14 complexand the subsequent depletion of PI(3,5)Ptriggers a multifaceted compensatory stress response that couples lysosomal proteostasis with mitochondrial bioenergetics

Key mechanisms include: Fat Oxidation: Facilitates the transport of fatty acids into the mitochondria for oxidation and conversion into usable energy, potentially enhancing fat-burning

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