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As a result, oxidized glucose products containing hydroxyl and carboxyl groups can form hydrogen bonds with residual oxygen functionalities on the GO surfaces to reduce to a stable glucose-rGO colloidal dispersion [78]
Therefore, erythrophagocytosis-induced ferroptosis contributes to the formation of larger atherosclerotic plaques, but this effect can be reversed by UAMC-3203 (Puylaert et al., 2023)
[DOI] [PubMed] [Google Scholar] 208.Moji M, Pristov JB, Maksimovi-Ivani D, Jones DR, Stani M, Mijatovi S, et al
They are not the same level of certainty
