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(a) Under normal conditions, apoC-III gene expression and synthesis are regulated by several factors, including nuclear transcription factors PPAR-, PPAR, Rev-erb, farnesoid X receptor, as well as insulin and glucose
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However, when bone marrow transplants from apoC1-KO or wild-type mice to apoE-KO mice were conducted to assess the specific effect of macrophage-derived apoC1 on the development of atherosclerosis, no difference was found for plasma either lipid levels or atherosclerotic lesions [128]
