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While modulating neurogenesis may provide a much-needed therapeutic opportunity of tackling post-stroke neurocognitive deficits, we lack a full understanding of the functional consequences of the increased neurogenesis following stroke, and of the mechanisms that mediate it
Researchers study NAD+ in the context of mitochondrial metabolism, oxidative stress models, and enzyme systems such as sirtuins and PARPs
The key to success lies in choosing an experienced provider, setting realistic expectations, and following pre and post treatment guidelines carefully
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