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N.SeminottiB.RibeiroC

The non-enzymatic mitochondrial protein hyperacetylation induced by the increased availability of mitochondrial acetyl-CoA avoids the risk associated with mitochondria overfeeding, restraining the activity of the respiratory complex I and modulating CACT activity

10.1111/j.1471-4159.2010.06807.x 26

The most common approach is to employ specific inhibitors to dissect/hinder the function of gB, gH, and gD in the entry process [117]
