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Description
It is most commonly treated with immunosuppressants and acetylcholine esterase inhibitors [22]
In neurodegenerative diseases, TMAO penetrates the BBB, impairing synaptic plasticity through PERK activation or mTOR pathway inhibition (Zhou et al., 2023), while promoting A and tau protein aggregation in Alzheimers and Parkinsons disease progression
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These findings suggest that early delivery of antioxidant gene therapy, such as AAV1-Sod2, could help preserve retinal function and prevent the progression of dry AMD [162]
