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Using ultra-sensitive proteomics, researchers in Maryland develop an accelerated-aging Alzheimers disease model and find that aging reshapes signaling pathways

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This remnant is rapidly taken up by the liver by a process that involves an initial sequestration of remnant particles on hepatic cell surface proteoglycans, followed by receptor-mediated endocytosis of remnants through the interaction of apoE with the chylomicron remnant receptor (LRP) (also called the low density lipoprotein receptor-like protein) or with the LDLR on the surface of hepatic parenchymal cells (10) (Fig 1.1)