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Description
In a rat model of hepatic ischemia reperfusion injury whereby cellular damage is caused by the restoration of oxygen delivery to a hypoxic liver pre-treatment with sulforaphane limited the reduction in glutathione (GSH) and the antioxidant enzymes, superoxide dismutase (SOD) and GSH peroxidase (GPx)

Both case control and cohort studies were excluded

The steps are reversible, with the equilibrium shifted toward product by the final deprotonation of the carboxylic acid

Known as the "master antioxidant," glutathione plays a critical role in neutralizing free radicals, supporting cellular repair, and maintaining overall balance in the body
