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Poor methylation can therefore lead to imbalances in neurotransmitter synthesis and breakdown, affecting a person's mental health, motivation, and energy levels

Tumor-specific targeting by IB-DNQ and Rucaparib combination treatment provides strong support for their potential use as a clinical therapy for patients with TNBC and our findings provide a compendium of cellular signaling changes with short single-agent and combination treatments defining the pathways that are highly affected that could contribute to cancer resistance mechanisms