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Collectively, HDL and apoA-I inhibit circulating levels of hematopoietic progenitor cells, monocytes, neutrophils, and platelets all of which contribute to HDLs capacity to limit inflammation and atherosclerosis

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Natural and synthetic inhibitors of Pgp [10, 11], liposomal formulations [12], co-delivery of Pgp inhibitors plus doxorubicin [13], have been tested to reduce Pgp activity in vitro and in preclinical models, but until now none of these approaches were effective in patients